carbon-nanotube是什么意思
carbon-nanotube
碳纳米管
词典结果：
carbon
[英][ˈkɑ:bən][美][ˈkɑ:rbən]
n.[化学]碳; （一张）复写纸; [电]碳精棒[片，粉]，碳精电极; 复写的副本;
adj.碳的; 碳处理的;
复数：carbons
易混淆单词：Carbon
以上结果来自金山词霸
例句:
1.
Exhortations to cut carbon emissions produce the same dichotomy.
要求中国减少碳排放也同样产生了两极分化的效果。
carbon nanotubes是什么意思
1. 碳纳米管
2. 纳米碳管
3. 奈米碳管
4. 巴基管
例句:
1.
Some researchers are working on sponges made of carbon nanotubes thatsoak up hydrogen.
一些研究者正在致力于由碳纳米管制成的海绵体来吸收氢气。
2.
Others, made of iron phosphate covered in carbon nanotubes, form thepositive poles.
一些则由磷酸铁制成，由碳纳米管包裹，作为电池的正极。
carbon-nanotube是什么意思
carbon-nanotube
碳纳米管
词典结果：
carbon
[英][ˈkɑ:bən][美][ˈkɑ:rbən]
n.[化学]碳; （一张）复写纸; [电]碳精棒[片，粉]，碳精电极; 复写的副本;
adj.碳的; 碳处理的;
例句:
Exhortations to cut carbon emissions produce the same dichotomy.
要求中国减少碳排放也同样产生了两极分化的效果。
multi-walled carbon nanotubes是什么意思
multi-walled carbon nanotubes
和多壁式纳米碳管；和多壁式奈米碳管；多壁奈米碳管
例句
1.Studies on Fluorescent Properties of Multi-Walled Carbon Nanotubes before and after Concentrated Nitric Acid Treatment
浓硝酸处理前后多壁碳纳米管的荧光特性研究
2.Effects of Gas Flow Rate on the Preparation of Multi-walled Carbon Nanotubes Using Chemical Vapor Deposition
气体流量对化学气相沉积法制备碳纳米管的影响
3.Co-Mo-K Sulfide-Based Catalyst Promoted by Multi-walled Carbon Nanotubes for Higher Alcohol Synthesis from Syngas
碳纳米管促进的Co-Mo-K硫化物基催化剂用于合成气制低碳混合醇
4.In this paper we investigate the effect of intershell coupling effect to nonlinear photoluminescence in multi walled carbon nanotubes.
研究了耦合效应对多壁纳米碳管非线性光致发光的影响。
5.Purification and Preliminary Modification of Multi-walled Carbon Nanotubes
多壁碳纳米管的纯化及修饰
多壁碳纳米管的特性
碳纳米管的独特结构决定了它具有许多特殊的物理和化学性质。组成碳纳米管的 C=C 共价键是自然界最稳定的化学键，所以使得碳纳米管具有非常优异的力学性能。理论计算表明，碳纳米管具有极高的强度和极大的韧性。其理论值估计杨氏模量可达 5TPa，强度约为钢的 100 倍，而重量密度却只有钢的 1/6。Treacy 等首次利用了 TEM 测量了温度从室温到 800 度变化范围内多壁碳纳米管的均方振幅，从而推导出多壁碳纳米管的平均杨氏模量约为 1.8Tpa。而 Salvetat 等测量了小直径的单壁碳纳米管的杨氏模量，并导出其剪切模量为 1Tpa。Wong 等用原子力显微镜测量多壁碳纳米管的弯曲强度平均值为 14.2±10.8GPa，而碳纤维的弯曲强度却仅有 1GPa。碳纳米管无论是强度还是韧性，都远远优于任何纤维，被认为是未来的“超级纤维”。
多壁碳纳米管的简介
1985 年英国萨塞克斯大学的波谱学家 Kroto 教授与美国莱斯大学的 Smalley和 Curl 两教授在合作研究中，发现碳元素可以形成由 60 个或 70 个碳原子构成的高度对称性笼状结构的 C60和 C70分子，被称为巴基球（Buckyballs）。1991 年，日本 NEC 科学家 Iijima 在制取 C60的阴极结疤中首次采用高分辨隧道电子显微镜发现一种外径为 515nm、内径为 213nm，仅由两层同轴类石墨圆柱面叠而成的碳纳米管。随后在 1993 年，Iijima和 Bethune研究小组同时报道合成了结构十分简单的单壁碳纳米管，这为理论预测碳纳米管的性能提供了实验上的可能性，进一步拓宽了碳簇材料的范围，也极大地促进了对碳纳米管的理论和实验研究，使得该领域成为如今全球研究的一个热点 。
翻译 中译英
Male mice in the AOM/DSS model were randomly divided into 4 groups: Model group, Model+AAV-NC group, Model+AAV/NC+BBD group, and Model/OE-SNTB1+BBD group. After one week of adaptive feeding, mice were injected with 12.5mg/kg AOM. After one week, they were given 2.5% DSS to induce the construction of colitis-associated colon cancer (CAC) model. SNTB1 gene overexpression plasmid was packaged and recombinant virus particles were obtained. The colon tissue of the mice was infected with the virus particles. At the beginning of each round of DSS, mice in the Model+AAV-NC and Model+AAV/NC+BBD groups were injected with 100ul of empty adenovirus, while mice in the Model/OE-SNTB1+BBD group were injected with 100ul of SNTB1 overexpression adenovirus. After the first round of DSS feeding ended, mice were given BaBaoDan (250mg/kg) for long-term intervention. Tissue analysis was conducted on day 67 of the experiment. The general living conditions of the mice and their weight were observed. An in viv【摘要】翻译 中译英【提问】我这边已经机翻好了，需要您帮我看看语法什么的，检查和更改一下【提问】亲 发来看看呢【回答】发文字哦【回答】中文摘要目的：通过体内外实验，在体外水平研究八宝丹（BaBaoDan，BBD）治疗大肠癌细胞的作用机制，检验SNTB1是否是八宝丹发挥治疗大肠癌功能的潜在靶点；以及在体内水平研究检验八宝丹是否通过调控SNTB1的表达，影响p38MAPK信号通路活化，参与调控结肠炎相关性肠癌（CAC）的发生发展，从而为临床应用八宝丹治疗CAC奠定基础,也为结肠炎相关性结肠癌的治疗提供了基因治疗新的思路。方法：1.体外培养SNTB1、LOVO肠癌细胞，采用慢病毒感染和嘌呤霉素筛选构分别构建大肠癌稳转细胞株，将2株细胞分为HCT-116/NC组、HCT-116/OE组及LOVO/NC组、LOVO/OE组；采用MTT法、细胞克隆球形成实验、Transwell、Invasion实验分别检测转染后各组细胞的活力、增殖、迁移侵袭能力； qPCR法检测转染后各组SNTB1的表达情况；WB检测过表达组细胞中SNTB1的表达是否被上调以及MAPK12、PMAPK12表达能力。【提问】AbstractObjective: To investigate the mechanism of BaBaoDan (BBD) in the treatment of colorectal cancer cells in vitro and in vivo, and to test whether SNTB1 is a potential target for BaBaoDan to exert its therapeutic effect on colorectal cancer cells; "And in vivo level studies to test whether Babao Dan can regulate the expression of SNTB1, affect the activation of p38MAPK signaling pathway, and participate in the regulation of the occurrence and development of colitis associated colon cancer【提问】Abstract:The objective of this study is to investigate the mechanism of BaBaoDan (BBD) in treating colorectal cancer (CRC) cells in vitro and in vivo, and to verify whether SNTB1 is a potential target for the therapeutic effect of BBD on CRC. The study also aims to examine whether BBD can affect the activation of p38MAPK signaling pathway by regulating the expression of SNTB1 in vivo and participate in the regulation of the occurrence and development of colitis-associated colon cancer (CAC), thus laying a foundation for the clinical application of BBD in the treatment of CAC and providing a new approach to gene therapy for colitis-associated colon cancer.Methods:SNTB1 and LOVO CRC cells were cultured in vitro, and stable cell lines of CRC were constructed by lentivirus infection and puromycin screening. The two cell lines were divided into HCT-116/NC group, HCT-116/OE group, LOVO/NC group, and LOVO/OE group. MTT assay, cell clone sphere formation experiment, Transwell, and Invasion【回答】(CAC), thereby laying the foundation for the clinical application of BBD in the treatment of CAC and providing a new gene therapy approach for colitis-associated colon cancer.Methods: SNTB1 and LOVO colorectal cancer cells were cultured in vitro, and stable cell lines were constructed using lentivirus infection and puromycin screening. The cells were divided into HCT-116/NC, HCT-116/OE, LOVO/NC, and LOVO/OE groups. The cell viability, proliferation, migration, and invasion ability of each group after transfection were assessed using MTT assay, cell clone sphere formation experiment, Transwell, and Invasion assay. qPCR was used to detect the expression of SNTB1 in each group after transfection, and WB was used to detect whether the expression of SNTB1 was upregulated and the expression ability of MAPK12 and PMAPK12 in the overexpressed cells.【回答】【提问】【提问】亲 这个太长了吧，8毛钱很难做到啊[流泪]【回答】好的，感谢【提问】我慢慢来吧，一段一段发给您【回答】Objective: To investigate the mechanism of BaBaoDan (BBD) in the treatment of colorectal cancer cells in vitro and in vivo, and to verify whether SNTB1 is a potential target for BBD to exert its therapeutic effect on colorectal cancer cells. Additionally, the study aims to examine whether BBD can affect the activation of p38MAPK signaling pathway by regulating the expression of SNTB1 in vivo and participate in the regulation of the occurrence and development of colitis-associated colon cancer (CAC), thus laying a foundation for the clinical application of BBD in the treatment of CAC and providing a new gene therapy approach for colitis-associated colon cancer.Methods: SNTB1 and LOVO colorectal cancer cells were cultured in vitro, and stable cell lines were constructed using lentivirus infection and puromycin screening. The cells were divided into HCT-116/NC, HCT-116/OE, LOVO/NC, and LOVO/OE groups. The cell viability, proliferation, migration, and invasion ability of each group after【回答】中文摘要目的:通过体内外实验，在体外水平研究八宝丹(BaBaoDan，BBD)治疗大肠癌细胞的作用机制检验SNTB1是否是八宝丹发挥治疗大肠癌功能的潜在靶点;以及在体内水平研究检验八宝丹是否通过调控SNTB1的表达，影响p38MAPK信号通路活化，参与调控结肠炎相关性肠癌(CAC)的发生发展，从而为临床应用八宝丹治疗CAC奠定基础也为结肠炎相关性结肠癌的治疗提供了基因治疗新的思路。方法:1体外培养SNTB1、LOVO肠癌细胞，采用慢病毒感染和漂岭霉素筛选构分别构建大肠癌稳转细胞株，将2株细胞分为HCT-116/NC组、HCT-116/OE组及LOVO/NC组、LOVO/OE组;采用MTT法、细胞克隆坏成实觉Iranswellnvasion:染后各组细胞的活力、增殖、迁移侵袭能力:qPCR法检测转染后各组SNTB1的表达情况:WB检测过表达组细胞中SNTB1的表达是否被上调以及MAPK12PMAPK12表达能力。2.将构建成功后的大肠癌稳转细胞株采用MTT法、细胞克隆球形成实验、Transwell、invasionScratch实验分别检测转染后各组细胞的活力、增殖迁移侵袭、转移能力:qPCR法检测转染后各组SNTB1的表达情况WB检测过表达组细胞中SNTB的表达是否被上调以及MAPK12、PMAPK12表达能力，以检测八宝丹对SNTB1介导的大肠癌细胞模型的影响。【回答】这段的【回答】SNTB1 adenovirus was constructed, and male Balb/c mice were randomly divided into 5 groups: Model group, Model/OE-NC group, Model/OE group, Model/Sh-NC group, and Model/Sh group. The general living conditions of the mice, as well as their weight and fecal occult blood were observed. The virus infection of the colon tissue was observed using an in vivo imaging system. The virus infection effect was verified using frozen sections, and the expression of SNTB1, MAPK12, and PMAPK12 in colon tissue was detected using IHC. RT-PCR was used to detect the differences in SNTB1 mRNA expression in colon tissue among the groups of mice, and Western blot was used to detect the expression of SNTB1, MAPK12, and PMAPK12.【回答】3构建SNTB1腺相关病毒，将雄性Balb/c小鼠随机分成5组，Model组、ModeVOE-NC组Model/OE组、Model/Sh-NC组、Model/Sh组;观察小鼠一般生活状态和测量体重、粪便隐血情况，通过活体成像系统观察结肠组织病毒感染情况，冰冻切片验证病毒感染效果，通过IHC检测结肠组织SNTB1、MAPK12PMAPK12表达;利用RT-gPCR技术检测各组小鼠的结肠组织SNTB1mRNA表达差异，通过Westermn Blot检测SNTB1、MAPK12、PMAPK12表达。这段的【回答】Male mice in the AOM/DSS model were randomly divided into 4 groups: Model group, Model+AAV-NC group, Model+AAV/NC+BBD group, and Model/OE-SNTB1+BBD group. After one week of adaptive feeding, mice were injected with 12.5mg/kg AOM. After one week, they were given 2.5% DSS to induce the construction of colitis-associated colon cancer (CAC) model. SNTB1 gene overexpression plasmid was packaged and recombinant virus particles were obtained. The colon tissue of the mice was infected with the virus particles. At the beginning of each round of DSS, mice in the Model+AAV-NC and Model+AAV/NC+BBD groups were injected with 100ul of empty adenovirus, while mice in the Model/OE-SNTB1+BBD group were injected with 100ul of SNTB1 overexpression adenovirus. After the first round of DSS feeding ended, mice were given BaBaoDan (250mg/kg) for long-term intervention. Tissue analysis was conducted on day 67 of the experiment. The general living conditions of the mice and their weight were observed. An in viv【回答】4.将AOM/DSS模型小凰随机分成4组，分别是Model组、Model+AAV-NC组Model+AAV/NC+BBD组、Model/OE-SNTB1+BBD组，经适应性喂养一周后，对小鼠体内注射12.5mg/kg的氧化偶氮甲烷(AOM)，一周后，给予2.5%浓度的葡聚糖硫酸钠(DSS)，诱导构建结肠炎相关性结肠癌(CAC)模型。将构建的SNTB1基因过表达质粒包装、重组得到病毒颗粒，感染小鼠结肠组织部位，在每一轮DSS开始时，Model+AAV-NC组及Model+AAV/NC+BBD组小鼠注射100ul/只的空载腺病毒，Model/OE-SNTB1+BBD组小鼠注射100ul/只的SNTB1过表达腺病毒。在第一轮DSS喂养结束后odeltAAWNCRRDEB1+RRD6小鼠使用八宝丹(250mg/kg)长期干预:在实验第67天的时候进行取材分析。观察小鼠一般生活状态和检测体重，运用小动物活体成像系统观察结肠组织病毒感染情况;利用冰冻切片验证病毒感染效果，免疫组化检测SNTB1、MAPK12、PMAPK12阳性率评价长期使用八宝丹对脏器组织病理学的影响，HE染色检测结肠组织瘤体情况，RT-GPCR检测结肠组织SNTB1表达差异;Western Blot检测SNTB1蛋白、p-MAPK12/MAPK12通路的表达情况，以验证八宝丹是否是通过调控SNTB1基因影响结肠炎相关性结肠癌小鼠的发生发展。这段的【回答】Results:The MTT experiment showed that overexpression of the SNTB1 gene can enhance the viability of HCT116 and LOVO cells (P<0.01). Compared with the HCT116/NC and LOVO/NC groups, the viability of cells in the HCT-116/OE and LOVO/OE groups was significantly increased. The colony formation experiment showed that overexpression of the SNTB1 gene can significantly promote the sphere-forming ability and proliferation of colon cancer cells. Compared with the HCT-116/NC and LOVO/NC groups, the cloning ability of cells in the HCT-116/OE and LOVO/OE groups was significantly improved, with significant differences (P<0.01). The Transwell and Invasion Scratch experiments showed that the migration, invasion, and scratch healing rate of HCT-116/OE and LOVO/OE colon cancer cells were higher than those of the HCT-116/NC and LOVO/NC groups (P<0.01), indicating that overexpression of the SNTB1 gene can promote the metastasis of colon cancer cells. qPCR detected the expression of SNTB1 in each group【回答】结果:1MTT实验表明SNTB1基因过表达能增强HCT116及LOVO细胞的活力(P<0.01)，与HCT116/NC组和LOVO/NC组相比，HCT-116/OE组与LOVO/OE组细胞的活力显著增加;集落形成实验表明SNTB1基因过表达能显著促进肠癌细胞成球能力与增殖情况，与HC-116/NC组和LOVO/NC组与LOVO/OE组细胞的克隆球形成能力显著提高，结果具有显著性差异(P<0.01)。Transwell、InvasionScratch实验表明转染后的HCT-116/OE组LOVO/OE组肠癌细胞其迁移侵袭及划痕应合率都高于HCT-116/NC组和LOVO/NC组(P<0.01)，其结果表明过表达SNTB1基因可促进大肠癌细胞的转移。qPCR法检测转染后各组SNTB1结果显示，与HCT-116/NC组和LOVO/NC组相比，HCT116/OE组及LOVO/OE组肠癌细胞的SNTB1的mRNA表达量显著增加(P<0.01)。Western blot结果显示，与HCT116/NC组和LOVO/NC组相比，HCT116/OE组及LOVO/OE组肠癌细胞的SNTB1的蛋白表达量及pMAPK12/MAPK12的比值显著升高(P<0.01)。2.经过八宝丹干预后，MTT实验结果显示，与HCT-116/NC组及LOVO/NC组的细胞相比，HCT116/SNTB1组及LOVO/SNTB1组的肠癌细胞的增殖能力显著提高(P<0.01)HCT-116/NC+BBD组与LOVO/NC+BBD组的细胞增殖水平降低(P<0.01)。八宝丹干预过后，HCT-116/SNTB1+BBD组及LOVO/SNTB1+BBD组的肠癌细胞增殖能力有所下降(P<0.05)这些结果表明八宝丹能通过影响SNTB1基因表达水平，降低肠癌细胞的增殖情况。这段的【回答】The Transwell, Invasion, and Scratch experiments showed that after intervention with BaBaoDan, the migration, invasion, and scratch healing rate of HCT116/NC and LOVO/NC colon cancer cells were significantly decreased (P<0.01), indicating that BaBaoDan can inhibit the metastasis of colon cancer cells. Additionally, compared with HCT116/SNTB1 and LOVO/SNTB1 overexpressing cells, the migration, invasion, and scratch healing rate of HCT116/SNTB1+BBD and LOVO/SNTB1+BBD colon cancer cells were also decreased (P<0.05). These results suggest that BaBaoDan can reduce the metastasis of colon cancer cells by affecting the expression level of the SNTB1 gene.In the qPCR experiment, it was found that the mRNA expression levels of SNTB1, MAPK12, and p-MAPK12 in HCT-116/NC+BBD and LOVO/NC+BBD groups were significantly decreased compared with the HCT-116/NC and LOVO/NC groups (P<0.05). Similarly, the mRNA expression levels of SNTB1 and MAPK12, as well as the protein expression level of SNTB1 and the【回答】TanswellInvasion、Scratch实验表明在HCT116/NCLOVO/NC组细胞中加入八宝丹的干预后，其迁移侵袭及划痕愈合率都较NC组细胞有明显的下降(P<0.01)其结果表明使用八宝丹可抑制大肠癌细胞的转移:同时，与HCT116/SNTB1及LOVO/SNTB1过表达细胞株相比，八宝丹使用后的HCT116/SNTB1+BBD与LOVO/SNTB1+BBD组肠癌细胞的迁移侵袭及划痕愈合率都有所下降(P<0.05)。这些结果表明八宝丹能通过影响SNTB1基因表达水平，降低肠癌细胞的转移在qPCR实验中，我们发现与HCT116/NCLOVO/NC组相比，HCT-116/NC+BBDLOVO/NC+BBD组的SNTB1、MAPK12、P-MAPK12Ln&rLOVO/SNTB1组相比，HCT116/SNTB1+BBD与LOVO/SNTB1+BBD组细胞中的SNTB1、MAPK12p-MAPK12mRNA表达水平下降，表明八宝丹可降低大肠癌细胞中的SNTB1、MAPK12、P-MAPK12等基因的mRNA表达水平(P<0.05)。Westemn blot结果显示，与HCT116/NC、LOVONC组相比，HCT116/NC+BBD、LOVO/NC+BBD的SNTB1蛋白表达量、P-MAPK12/MAPK12的比值下降(P<0.01);与HCT-116/SNTB1及LOVO/SNTB1组相比，HCT116/SNTB1+BBD与LOVO/SNTB1+BBD组细胞中的SNTB1蛋白表达量、P-MAPK12mRNA表达水平下降，表明八宝丹可能是通过介导SNTB1，进而发挥调控p38MAPK信号通路，抑制大肠癌发生发展的作用。这段的【回答】Compared with the Control group, the Model group of mice showed a decline in their living conditions (including fur luster, activity, and diet), and the Model/OE group with SNTB1 overexpression exhibited significant weight loss, bloody stool, and diarrhea (P<0.001). However, the severity of the disease in the Model/Sh group with SNTB1 knockdown was not significant, indicating that SNTB1 may be a potential target gene that promotes the development of colitis-associated colorectal cancer. The small animal live imaging results showed that after injection of adenovirus for 2-3 weeks, the colon tissues of the Model/OE-NC group, Model/OE group, Model/Sh-NC group, and Model/Sh group of mice showed strong fluorescence under the small animal live imaging system, indicating that the adenovirus had infected the colon tissues. The IHC staining results showed that compared with the Model group, the positive expression levels of SNTB1, MAPK12, and p-MAPK12 in the colon tissues of the Model/OE group【回答】3与Control组相比，Model组小鼠生活状态(皮毛光泽、活动状态、饮食等)下降，SNTB1过表达的Model/OE组小鼠出现明显的体重下降便血及腹泻(P<001)，而SNTB1敲减的Model/Sh组小鼠疾病严重程度则不明显，表明SNTB1可能是促进肠炎相关性肠癌发展过程中的潜在靶基因。小动物活体成像结果显示，在注射腺病毒2~3周后，Model/OE-NC组、Model/OE组、Model/Sh-NC组、Model/Sh组小鼠的结肠组织在小动物活体成像系统下观察可呈强荧光，说明腺病毒已感染了结肠组织。IHC染色结果发现，与Model组相比，Model/OE组结肠组织中SNTB1、MAPK12PMAPK12的阳性表达量明显升高(P<0.01)，而Model/Sh组SNTB1、MAPK12PMAPK12的阳性表达量降低。qPCR实验结果显示与Model组相比，Model/OE组SNTB1的mRNA表达量显著增高(P<0.05)，而Model/Sh中的SNTB1mRNA表达量显著降低(P<0.05)。Western blot结果显示，与Model组相比，SNTB1过表达组的SNTB1蛋白表达量、P-MAPK12/MAPK12的比值升高(P<0.01)而SNTB1敲减组的SNTB1蛋白表达量、PMAPK12/MAPK12的比值显著降低(P<001)。这段的【回答】The results of the study showed that Ba Bao Dan could inhibit the proliferation and metastasis of colorectal cancer cells by regulating the expression of SNTB1 and the p38MAPK signaling pathway in the cells, as well as relieve the disease activity index, improve the survival status and pathological condition of mice. SNTB1 may be an important target for Ba Bao Dan to inhibit the development of colitis-associated colorectal cancer. Keywords include Ba Bao Dan, SNTB1, p38MAPK12 signaling pathway, colitis-associated colorectal cancer, metastasis.After intervention with Ba Bao Dan, the results of the sample showed that compared with the Model+AAV-NC group, the disease activity of mice in the Model/NC+BBD group and the Model/OE-SNTB1+BBD group improved, with a decrease in severity, weight loss, and relief of rectal bleeding and diarrhea index (P<0.05). IHC staining results showed that compared with the Model+AAV-NC group, the number of SNTB1 MAPK12 and P-MAPK12 positive cells in the Model+【回答】4.加入八宝丹干预后，取材结果显示，与Model+AAV-NC组相比，Model/NC+BBD组及Model/OE-SNTB1+BBD组小鼠疾病活动情况有所改善，严重程度降低，体重下降、便血及腹泻指数有所缓解(P<0.05)。IHC染色结果发现，与Model+AAV-NC组相比，Model+AAV/NC+BBD组SNTB1MAPK12、P-MAPK12阳性细胞数量显著降低(P<001)，而Model/OE-SNTB1+BBD(P<005)组的SNTB1、MAPK12、p-MAPK12阳性率也有所下降(P<0.05)。HE染色结果显示，与Model+AAV-NC组相比，Model+AAV/NC+BBD组的小鼠结肠组织炎症细胞浸润的情况有所缓解，肠道组织结构更为完整(P<0.05)。qPCR实验结果显示，与Model+AAV-NC组相比，Model+AAV/NC+BBD组SNTB1的mRNA表达量显著下降(P<0.01);Model/OE-SNTB1+BBD组SNTB1的mRNA表达量有所下调(P<005)。Westernblot结果显示，与Model+AAV-NC组相比MAPK12/MAPK12的比值显著降低(P<0.01)而Model/OE-SNTB1+BBD组SNTB1蛋白表达量、pMAPK12/MAPK12的比值有所下降(P<005)。结论:SNTB1可能是八宝丹抑制结肠炎相关性结肠癌生成的重要靶标，过表达SNTB1能增强肠癌细胞的活力、增殖及转移能力，加重结肠炎相关性结肠癌小鼠的疾病严重程度。而使用八宝丹干预后，八宝丹可通过介导大肠癌细胞中SNTB1的表达，调控p38MAPK信号通路，进而发挥抑制大肠癌细胞增殖及转移的作用。同时八宝丹也可以靶向调节SNTB1的表达，缓解疾病活动指数，改善小鼠生存状态及病理情况;八宝丹也可以调控p38MAPK信号通路，从而发挥其抑制大肠癌转移和治疗大肠癌的目的。关键词:八宝丹，SNTB1，p38MAPK12信号通路，结肠炎相关性结肠癌，转移这段的【回答】亲 完结了哦【回答】
中英翻译
1.中英翻译可以分为英译汉和汉译英。英译汉的意思是英文翻译为中文。汉译英的意思是中文翻译成英文。翻译是在准确（信）、通顺（达）、优美（雅）的基础上，把一种语言信息转变成另一种语言信息的行为，让更多人了解其他语言的含义。翻译是将一种相对陌生的表达方式，转换成相对熟悉的表达方式的过程。其内容有语言、文字、图形、符号和视频翻译。翻译有口译、笔译、机器翻译、同声传译、影视译配、网站汉化、图书翻译等形式。
2.英语（英语：English）是一种西日耳曼语支，最早被中世纪的英国使用，并因其广阔的殖民地而成为世界使用面积最广的语言。英国人的祖先盎格鲁部落是后来迁移到大不列颠岛地区的日耳曼部落之一，称为英格兰。这两个名字都来自波罗的海半岛的Anglia。该语言与弗里斯兰语和下撒克森语密切相关，其词汇受到其他日耳曼语系语言的影响，尤其是北欧语（北日耳曼语），并在很大程度上由拉丁文和法文撰写。
3.英语已经发展了1400多年。英语的最早形式是由盎格鲁-撒克逊人移民于5世纪带到英国的一组西日耳曼语支（Ingvaeonic）方言，被统称为古英语。中古英语始于11世纪末，诺曼征服英格兰；1476年，威廉·卡克斯顿将印刷机介绍给英国，并开始在伦敦出版第一本印刷书籍，扩大了英语的影响力。自17世纪以来，现代英语在英国和美国的广泛影响下在世界各地传播。通过各类这些国家的印刷和电子媒体，英语已成为国际主导语言之一，在许多地区和专业的环境下的语言也有主导地位，例如科学、导航和法律。
麻烦大家帮我翻译下
爱占小便宜,终生难大贵；经常吃小亏,日久必厚报。2.言语多反复,当防欺诈；忘恩思小过,定会反戈。3.热情过度,必然另有名堂；严肃有(2/3)余,切勿敞开心扉。4.开口说大义,临大难必变节；逢人称兄弟,即深交也平常。5.谦为美德,过谦则防诈；默为懿行,过默则藏奸。6.揽功而推(3/3)过，不可同谋共事请问各位天才 麻烦你们帮我翻译一下这几句话的意思 谢谢
Being fond of taking small advantage can't be very successful and very prosperous all the life
To take frequent small loss must be rewarded tremendously some day in the future
麻烦帮我翻译一下
一个小男孩走在街上遇到了一条蛇。
这只蛇说“你能带我到山顶吗？我想在我死之前最后再看一次日落。”
而这个男孩说“不，亲爱的蛇先生。如果我捡起你，你就会咬我了。”
蛇又对男孩说“不，我永远都不会这么做。”于是这个男孩带着蛇去了山顶。
在日落后蛇又对男孩说“你能带我去你家吗？”男孩带着蛇回了家并给了它一些食物。
第二天，蛇对男孩说"请带我回到我的家，我是时候该离开这个世界了”
男孩认为他已经安全了，就带着蛇回到家。但是在他正准备放下蛇的时候，蛇反身咬了他。
男孩哭着说道“蛇先生，你为什么要这么做”
蛇大笑道“你不知道我是谁吗”
孔子的著作主要有哪些？
古人认为孔子曾修《诗》《书》《礼》《乐》，序《周易》（称《易经》十翼，或称易传），撰《春秋》。诗歌：《去鲁歌》、《蟪蛄歌》、《龟山操》、《盘操》、《猗兰操》、《将归操》、《获麟歌》。这些诗歌多载于《乐府诗集》，可信度不高拓展资料：孔子（公元前551年9月28日―公元前479年4月11日） ，子姓，孔氏，名丘，字仲尼 ，春秋末期鲁国陬邑人（今山东曲阜），祖籍宋国栗邑（今河南夏邑） 。孔子是中国古代著名思想家、教育家，他开创了私人讲学的风气，倡导仁、义、礼、智、信，是儒家学派创始人。道德思想孔子建构了完整的“德道”思想体系：在个体层面主张“仁、礼”之德性与德行。[30] 德道思想体系是以性善论（“一阴一阳之谓道，继之者善也，成之者性也”）为基础，以立人极（“三极之道”）为旨归，以人道与天道、地道相会通，人道中庸又适时之变为方法论的完足思想体系。孔子的仁说，体现了人道精神，孔子的礼说，则体现了礼制精神，即现代意义上的秩序和制度。人道主义这是人类永恒的主题，对于任何社会，任何时代，任何一个政府都是适用的，而秩序和制度社会则是建立人类文明社会的基本要求。孔子的这种人道主义和秩序精神是中国古代社会政治思想的精华。孔子晚年时期的最高理想称之为“大同”，在大同的世界里，天下的人，不止以自己的家人为亲，不止以自己的父母儿女为爱，而是相互敬爱，爱天下所有的人。使老有所终，壮有所用，孩子们都能获得温暖与关怀，孤独的人与残疾者都有所依靠，男人各自有自己的事情，女人有满意的归宿。天下没有欺诈，没有盗贼，路不拾遗，夜不闭户，人人讲信修睦，选贤举能，大道之行也，天下为公。政治思想孔子的政治思想核心内容是“礼”与“仁”，在治国的方略上，他主张“为政以德”，用道德和礼教来治理国家是最高尚的治国之道。这种治国方略也叫“德治”或“礼治”。这种方略把德、礼施之于民，严格了等级制，把贵族和庶民截然划分治者与被治者。打破了贵族和庶民间原有的一条重要界限。孔子生活在西周宗法礼制传统较深的鲁国，这时周王朝的统治权力已经名存实亡，诸侯间相互争战不断，出现了“王道哀，礼义废，政权失，家殊俗”的社会现实，“君不君、臣不臣、父不父、子不子”成了那个时代的特点。社会矛盾的激化阻碍了生产力的发展，人的精神和信念也发生了前所未有的摧残。这些共同构成了孔子政治思想产生的历史渊源和社会条件，“仁”与“礼”是其政治思想的基本精神。孔子的最高政治理想是建立“天下为公”的大同社会。“大同”社会的基本特点是：大道畅行，“天下为公”，因而能“选贤与能，讲信修睦”，“人不独亲其亲，不独子其子，使老有所终，壮有所用，幼有所长，矝寡孤独废疾者皆有所养”，阴谋欺诈不兴，盗窃祸乱不起，这是一幅理想化的传说中的尧舜时代的原始社会景象，也是孔子憧憬的最高理想社会。“小康”社会是孔子主张的较低的政治目标。“小康”社会的基本特点是：大道隐没，“天下为家”，“各亲其亲，各子其子，货力为己”，与这种贫富不均、贵贱不等相适应，产生了一系列的典章制度、伦理道德，“以正君臣，以笃父子，以睦兄弟，以和夫妇”，“以立田里，以贤勇知”，相应地还要设“城郭沟池以为固”，由是，“谋用是作，而兵由此起”。这种社会显然没有“大同”世界那样完美，但有正常秩序，有礼、仁、信、义，所以称为小康。这种社会实际上描述了“私有制”产生后的阶级社会的“盛世”。孔子的“大同”社会、“小康”社会理想对中国后世影响深远。后来不同历史时期，不同阶段的思想家提出不同内容的憧憬蓝图和奋斗目标，这种思想对进步思想家、改革家也有一定启发，洪秀全、康有为、谭嗣同和孙中山都受到其影响。历史地位西汉中期，天灾频发，社会动乱。儒者梅福认为，这是由于未能妥善安排对于孔子的祭祀，因而上天发怒。当时的国家政权接受了梅福的提议，封孔子为商汤的后代，接续先王的祭祀。到东汉，国家才正式把孔子作为国家的公神，其地位和社稷神同等。唐代，命令每个县都要建庙祭祀孔子。每年春秋两次大祭，每月初一和十五两次小祭。大的祭祀起初由学官主持，后来改由地方官主持。唐代以后，孔子的地位不断提高，对孔子的封号也不断增加。清代，孔子祭祀一度成为和上帝、和国家的祖宗神同等级别的“大祀”。这种殊荣除老子外万古唯有孔子而已。孔子成为国家公神以后，儒者们的宗教地位也相应提高。唐代为孔子设立“从祀”即陪同享受祭祀的制度（如道家四大真人）。最早选中陪同孔子的，是22位对于注释儒经有重大贡献的儒者，后来扩大到孔子的所有弟子和历代著名的儒者。宋代，从祀制度逐渐完备起来。其最高的有四位，被称为“四配”，他们是颜回、曾参、子思和孟轲。其次是“十哲”，孔子的十个优秀弟子。“德行：颜渊，闵子骞，冉伯牛，仲弓。言语：宰我，子贡。政事：冉有，季路。文学：子游，子夏。”再次是“先贤”，祭祀那些亲自接受孔子教导的弟子们。最后是“先儒”，祭祀孔子弟子以后历代最优秀的儒者。而后来的儒者，也以死后能够进入孔庙成为先儒为最高的荣誉。参考资料：孔子——百度百科
细胞成像多功能检测仪c3可以测荧光强度吗
荧光探针应用细胞成像，怎样找活体细胞为了提高活体细胞的显微图像分辨率虽然有多种方法,然而至今还未找到一种公认的处理所有活体细胞图像方法。本文采用伪彩色处理与多种图像处理方法结合处理50幅原始图像,并比较处理前后的卵母细胞图像,使用SPSS10.0统计软件包加以处理,探讨处理活体细胞是否能提高图像的分辨率及系统是否适用于活体细胞的图像处理,为今后临床和科研研究活体细胞创造条件。
荧光显微镜拍照为什么一定要加盖玻片
1、定位没问题，用图像处理就可以。一般来说要在同一个视野用明场（HE染色，或者苏木素复染）和荧光场同时拍照，然后用叠加就可以了。这个功能在photoshop上也可以实现，只要photoshop用的熟。
2、定量这个问题就相对差一些了，只能通过色度来分析，最好选择配套的荧光分析，当然也可以用photoshop来做（稍微麻烦一些）。但是这样得到的结果只能算是半定量的（数据处理多用秩和检验）。最好的办法还是荧光标记后的细胞过流式细胞仪，这样结果才最为信服。
